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Reprogramming of the FOXA1 cistrome in treatment-emergent neuroendocrine prostate cancer

By enhancing fat oxidation capacity and improving insulin sensitivity, 5 amino 1mq helps restore this metabolic flexibility

Notably, inhibition of MAPK/ERK signalling exacerbates 3-HK-induced mitochondrial dysfunction and cell death, implicating impaired cellular resilience pathways in amplifying KYN-mediated toxicity

Actual results may vary

Funding information This work was supported by National Science and Technology Council grant 101-2311-B-001-030-(to SCF)

Banhegyi G., Lusini L., Puskas F., Rossi R., Fulceri R., Braun L., Mile V., Simplicio P., , Mandl J., Benedetti A