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Conversely, sustained ferroptosis activation in the post-AKI phase drives tubular atrophy and interstitial fibrosis, transitioning adaptive repair to maladaptive progression

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Dendrimer-based systems, in particular, have been explored as scaffolds for adjuvant presentation and controlled immunomodulation [208]

The last category included MARS, SESN2, CHAC1, and FGF21, and their basal expression was also reduced by Erk1/2 inhibition

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