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The SSP is aberrantly activated in a variety of malignant tumors and promotes tumor progression through multi-dimensional mechanisms

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This results in an increase in the stability of N-calmodulin and a decrease in the stability of -catenin through the process of ubiquitination

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TNBC-2 shows upregulation in lipid metabolism, including cholesterol metabolism, estrogen metabolism, linoleate metabolism, arachidonic acid metabolism, omega-3 fatty acid metabolism, fatty acid biosynthesis, and glycolysis

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Then the glutathione becomes depleted and we can no longer protect ourselves against free radicals, infections, or cancer and we cant get rid of toxins

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