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McConnell JD, Roehrborn CG, Bautista OM, Andriole Jr GL, Dixon CM, Kusek JW, et al

pylori infection and CagA have been found to suppress ferroptosis, while erastin and RSL3 are capable of inducing ferroptosis in the treatment of GC infected with H

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Several other factors also contribute, including excess sebum production, hormonal changes & lifestyle

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Notably, these findings collectively highlight that NPC ferroptosis is a confirmed promotor of IDD by impairing the viability of NP-residing cells as well as damaging the extracellular matrix (ECM) 7,8 , while the operation-induced physical and biochemical insults could significantly promote the ferroptosis cascades in NPCs to enhance the degenerative phenotype as well as aggravate the IDD symptoms