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This might possibly be due to the engineered control circuits responding to the target product intermediate, limiting any improvement in production beyond initial control levels

This dysfunction exerts a profound influence on both bioenergetic metabolism and non-energetic pathogenesis [18]

Emerging perspectives in proteostasis: bridging mechanisms and therapeutics for human diseases

A simplified schematic illustration of the progression from simple hepatic steatosis to cirrhosis is presented in Figure 2

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Here we identified Lys105 of ScGrx7 and Lys26 of PfGrx as a dual activator for the catalytic cysteine residue and GSH