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TMAU is caused by gene mutations in the FMO3 gene, which is located on the long arm of chromosome 1 [2,13]

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The treatment of Alms1 -/- mice with mAb involved two approaches: (i) curative, consisting in the treatment of 20-week-old mice with an established AS phenotype for 4 weeks (Supplementary Fig

Parthenolide has been shown to inhibit the activation of the NLRP3 inflammasome.[ref] Plasmalogens (NLRP3, TNF): Plasmalogens are a unique type of phospholipid and an essential component of cell membranes