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Rodrguez-Espinosa et al., 2015

Mechanistically, targeting CD38 improved T cell glucose metabolism and mitochondrial mass in vitro, attenuated hippocampal glucose hypometabolism, as well as cortical IL-1-driven inflammation in vivo, an AD hallmark associated with disease progression 71,72 , linking metabolism and immunity 73

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3.2 SPR analysis of the interaction between VPNHFNAP and MPO protein To further validate the molecular simulation results and clarify the biological activity of VPNHFNAP, a SPR experiment was conducted using purified human MPO receptor protein and synthetic peptide VPNHFNAP