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The anemia caused by hematopoietic Gpx4 -deficiency shares a number of features with -thalassemia, for which the term ineffective erythropoiesis has been coined: decreased RBC, elevated reticulocyte counts, overactive extramedullary erythropoiesis, elevated erythroid progenitors, absence of hemolysis, and systemic iron overload linked to severe iron demand for the erythroid system.51 This prompted us to study iron metabolism in mice with Gpx4 -deficient hematopoiesis