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10.1016/j.numecd.2019.12.010 144 LaiX.-N.LiJ.TangL.-B.ChenW.-T.ZhangL.XiongL.-X

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and Klein, H.H

Activation of GPX4 partially rescued these effects, reinforcing the role of mGPDH as an auxiliary mitochondrial defense mechanism against ferroptosis under pathological stress

Agreement on clinically relevant outcome measures is also lacking,71 and many studies have clear conflicts of interest

Research Scope: Suitable for experimental studies involving peptide behavior and intracellular pathway analysis