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Figure 6A demonstrates that there was no detectable fluorescence signal at the tumor site 24 h after the intravenous administration of free RhB, suggesting that free RhB did not accumulate specifically at the tumor site

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New delivery mechanisms, such as microneedle patches, sustained release implants, and targeted liposomal carriers, are being developed to enhance tissue targeting and minimize dosing frequency

This impairment cripples the adaptive stress response, effectively lowering the threshold for oxidative damage and rendering airway structural cells markedly more vulnerable to ferroptosis upon encounter with other environmental triggers

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