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Researchers have found that high levels of homocysteine disrupt the integrity of the blood-brain barrier , which allows substances that are normally kept out of the brain to cross over and contribute to neurological problems (99-102)

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Several strategies have been proposed to alleviate this damage, including cell transplantation 4,5,6,7 and gene therapy 8,9,10,11

Once activated, SIRT1 deacetylates and activates PGC-1, the master transcriptional coactivator that directly commands the cell to build new mitochondria

elegans model but also in mammalian systems