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Ischemia-reperfusion injury in sickle cell anemia: relationship to acute chest syndrome, endothelial dysfunction, arterial vasculopathy, and inflammatory pain

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Therefore, considering the presence of APAP protein adducts at the therapeutic dose of APAP, formation of protein adducts on mitochondrial proteins rather than the overall formation of protein adducts may be the reason for cellular toxicity, which has been considered a key factor for necrotic cell death in previous years (Jollow et al., 1973

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Miwa, et al., Leukemia cells demonstrate a differen t metabolic perturbation provoked by 2-deoxyglucose

doi: 10.3389/fmolb.2021.671037 39 RussoEDi GloriaLCerboneschiMSmeazzettoSBaruzziGPRomanoFet al