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Throughout these evaluations, GHK appeared to lower ROS levels induced by tert-butyl hydroperoxide (t-BOPD), a chemical known to promote oxidative stress within cells

Recruitment of the p97 ATPase and ubiquitin ligases to the site of retrotranslocation at the endoplasmic reticulum membrane

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We propose that all considered effects of GPX4 on the risk of CS can be mediated by three following mechanisms

It is noteworthy that elevated concentrations of Epitalon (1 mM and 2 mM) did not substantially reduce ROS levels