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All participants provided written informed consent prior to any study procedures in accordance with the Declaration of Helsinki

Acta Neuropathol 132:197

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People with variants in the SLC22A5 gene (often referred to as carnitine transporter deficiency) which results in carnitine deficiency can have skeletal myopathy and/or cardiomyopathy [7]

BEA and enniatins are reported to be metabolized by carbonylation, carboxylation, hydroxylation, and N -demethylation based on experiments in vitro and in vivo in pigs