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The ability of G/D-CuP to scavenge reactive oxygen species (ROS) was evaluated by conducting Fenton-like reactions where hydrogen peroxide (H 2 O 2 ) generates reactive hydroxyl radicals (OH) with the help of metal catalysts, leading to significant oxidative harm 65

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In line with our hypothesis, raltitrexed also significantly increased mitochondrial content (Fig

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[DOI] [PubMed] [Google Scholar] 71.Bjelakovic G., Nikolova D., Gluud L.L., Simonetti R.G., Gluud C

As a consequence of AKI, a significant number of oxygen free radicals are produced, endogenous antioxidants are continually depleted, and high levels of inflammatory substances are secreted, all of which contribute to kidney damage progression ( According to our published papers ( Our previously single-center clinical studies indicated CHF might have nephroprotection in A on C patients ( Ligusticum wallichii (Chuanxiong) , has the potential to prevent AKI via a variety of processes, including ameliorating oxidative stress damage, suppressing inflammatory responses, deterring apoptotic cell death of intrinsic renal cells, and modulating autophagy ( Renal fibrosis is by far the most important mechanism that leads to CKD ( 1 is the primary profibrotic facilitator in kidney disorders due to its role as a key modulator of fibrosis ( 1 have additionally validated the profibrotic function of TGF- 1 in the etiology of progressive renal fibrosis in a variety of kidney illnesses ( 1 might be a possible treatment target for the clinical management of renal fibrosis