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Using microtubule staining, we observed that KU-57788 treatment caused hallmark features of mitotic catastrophe in ATC cells, including chromosomal misalignment and multipolar spindle formation during cell division (Fig

The potential mechanisms by which VPA manifests its antimycobacterial activity include NO production and histone deacetylase 1 (HDAC-1) inhibition ( Figure 3 )

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and (iv) Demonstrating that interventions based on such immune phenotyping can improve prognosis

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