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Ultimately, we advocate a rationale to improve the outcome of anti-MET therapies on the basis of thorough consideration of the entire spectrum of MET-mediated biological responses, which implicates adequate patient stratification, meaningful biomarkers and appropriate clinical end points

Targeting senescent cells alleviates obesityinduced metabolic dysfunction

The MetHGF interaction regulates various signalling pathways involving downstream kinases, such as Akt and Erk

Interplay between genetics and epigenetics in osteoarthritis

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