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Mechanistically, ipamorelin attenuates I/R-induced rises in hepatic MDA and 4-HNE through NRF2-HO-1-SOD2 induction, reduces caspase-3 and caspase-9 cleavage downstream of mitochondrial cytochrome c release, and suppresses NF-B-dependent TNF-, IL-1, and IL-6 production in Kupffer cells

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Essential role of excessive tryptophan and its neurometabolites in fatigue

The cellular derivation and the life span of the myofibroblast

This hypothesis, while speculative, could explain the peptides ability to normalize both deficiency and excess states across diverse neurotransmitter disturbances