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In human GB tissues, AATF+ cells exhibited lower levels of -H2AX than AATF cells (Fig

Macrophage activation and foam cell formation may explain the elevated CRP serum levels and contribute to disease progression (Figure 9)

Thiopurines themselves are inactive and require intracellular metabolism, catalyzed by multiple enzymes, to the active thioguanine nucleotides (TGN), responsible for causing immunosuppression [35]