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Furthermore, dihydroartemisinin has been demonstrated to increase the unstable iron pool, promote ROS accumulation, induce ferroptosis, and inhibit the proliferation of leukemia (HL-60, KG-1, and THP-1) cells [136]

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It further indicates that disrupting a specific link between the immune and coagulation systems may be conducive to the prevention and treatment of microvascular thrombotic lesions