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[PMC free article: PMC3546807] [PubMed: 23114962] 16

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Consistently, NIK-deficient blood monocytes display inflammatory phenotype in response to bacterial lipopolysaccharide (LPS) stimulation, and NIK-deficient mice display increased pro-inflammatory myeloid cells in the blood, bone marrow and adipose tissue, although it is unclear whether this phenotype is due to macrophage-intrinsic function of NIK [26]

These processes have implications not only on the survival of keratinocytes but also potentially influence the skins overall immune response by triggering the release of inflammatory mediators

Furthermore, the analysis indicated that, at 1 and 2 h following APAP administration, hepsin / mice displayed 156 and 141 differentially expressed genes, respectively, when compared with wild-type mice at the same time points