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Mitochondrial dysfunction arises a consequence of genomic instability, calcium ion overload, imbalanced redox reactions, dysregulation of mitochondrial turnover, and nutrient sensing pathways during the ageing process

Carbonylation of ER chaperone proteins in aged mouse liver

GSCs have a radio- and chemo-resistant phenotype responsible for the constant relapse (Liu et al., 2015), in part because of the high expression of anti-apoptotic proteins and drug efflux transporters (Nakai et al., 2009) and the constitutive activation of the DNA repair element, poly-ADP-ribose polymerase 1

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[DOI] [PubMed] [Google Scholar] 84.Stavri, M., Piddock, L

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