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doi: 10.1016/j.freeradbiomed.2021.07.007, 59

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FIGURE 2 Coincided with reduced AF inducibility, LCA supplementation showed significant capacity to shrink LA, attenuate cellular hypertrophy, gap junction disturbance, and fibrosis while challenged by a long-term HFD (Figure 2), indicating FAO promotion via LCA supplementation constrained the AF substrates in obese mice