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This phenomenon is due to the upregulation of the expression of glutamine transporters or enzymes by oncogenes such as c-MYC, which accelerate the uptake and metabolism rate of glutamine and then drive the TCA and biosynthetic processes [3, 16] (Fig

DHA inhibits AD pathogenesis by attenuating the A burden, inhibiting tau phosphorylation and decreasing neuroinflammation 98,99

Data shown in Fig

The homeostatic balance of these processes is crucial

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A large difference in basal concentrations of ergothioneine in whole blood was observed in a pharmacokinetic study by Cheah and colleagues (Reference Cheah, Tang and Yew61) , who also noted that participants with the highest basal levels of ergothioneine also appeared to take up more of the supplemented ergothioneine