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Glucose-6-phosphate will metabolically trifurcate into the following pathways: (i) conversion to fructose-6-phosphate by the enzyme phosphoglucose isomerase, entering the glycolytic pathway, or (ii) oxidation by glucose-6-phosphate dehydrogenase to 6-phosphogluconolactone, thereby initiating the oxidative phase of the pentose phosphate pathway

In sALS patients, deregulation of the cellular redox systems has been suggested by higher free radical levels found in the blood and serum of patients[32, 73], and evidence of oxidative damage includes the increase in protein carbonyls[34, 35, 74, 75], 8-OHdG[47, 49], MDA-modified proteins[34], 4-HNE protein conjugates[36, 76], and nitrotyrosine products[7779] in the spinal cord

In conclusion, the occurrence of DFU is closely related to cell dysfunction, low-grade inflammation, and tissue hypoxia induced by hyperglycemia

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MBio 11:19

The authors also identified increased activities in 15 metabolic pathways and decreased activities in 45 metabolic pathways in AMD patients compared to that of the controls (Xue et al., 2023)