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Of these, 160 articles were excluded because they did not address thyroid autoimmunity, thyroid outcomes, GLP-1RAs, or relevant immunometabolic pathways

Meanwhile, hypoxia and cell death caused by the expansion due to adipose tissue accumulation further recruit macrophages and promote the release of tumor necrosis factor-, interleukin-6, and interleukin-1, which induce inflammatory gene transcript in endothelial cells and suppresses endothelial NOS expression [156]

FSP1/CoQ10/NADPH system FSP1, identified as another ferroptosis suppressor through genome-wide screening, primarily prevents lipid peroxidation and ferroptosis by reducing lipid free radicals in lipid droplets or plasma membranes [112]

With this in mind, we functionalized MMF with a biologically compatible 1,2-dicarbonyl group (compounds 1ac

Jalalvand and Riesbeck, 2018

Stojsavljevi A, Rovanin B, Krsti et al (2019) Cadmium as main endocrine disruptor in papillary thyroid carcinoma and the significance of Cd/Se ratio for thyroid tissue pathophysiology