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Type V collagen: heterotypic type I/V collagen interactions in the regulation of fibril assembly

The results of this explicit experiment open an unexpected door revealing the ancient, complex roles of amyloids in all three major domains of life where the neurodegenerative effects of aberrant A aggregation may be the result of protective antimicrobial activity [81]

6p), significantly rescued the mitochondria fragmentation phenotype of ATG5-depleted LEC (Fig

the role of oxidative stress, apoptosis, and JNK1/2-P38 signaling pathways

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Finally, the in vivo mechanistic studies confirmed the modulation of MAPK and NF-B signaling pathways