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In this regard, a key mode of action to explain this relationship is via glucose transporter type 1 (GLUT1) which increases uptake of the oxidized form of ascorbic acid, dehydroascorbate and subsequently depletes glutathione (Yun et al., 2015)

& Ogawa, N

IF RT-PCR IF WB In Vivo Efficacy Study All Endogenous Metabolite Isoforms More Biological Activity [1] [2]

PERK activation enables tumor cells to transiently attenuate global protein synthesis while selectively enhancing the translation of stress-responsive genes that promote survival, invasion, and metabolic rewiring

Taken together, our findings indicate that upregulated SLC25A39 promotes CRC cell growth and migration by reducing ROS accumulation

3 5FU drives increases in mitochondrial content specifically in tumour cells