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In response to NO and peroxynitrite, MTs release Zn, which induces MTF-1, and this increases the expression of protective genes by scavenging some ROS and reactive nitrogen species [147]

trans-N-(2-Chlor-6-methylphenyl)-2-[[[2-[(dimethylamino)methyl]cyclopropyl]carbonyl]amino]-6-benzothiazolcarboxamid

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In multiple preclinical models, pharmacological inhibition of ferroptosis preserves myocardial and vascular function in settings such as ischemic injury, heart failure, and atherosclerosis, positioning ferroptosis as both a central disease driver and a potential therapeutic target

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This makes dual-agonist combination treatments possible