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Although no significant lipid peroxidation is observed in cancer-associated muscle atrophy, increased mitochondrial protein carbonylation impairs mitochondrial function, suggesting that regulating mitochondrial phospholipid biosynthesis pathways may be an effective therapeutic target for cancer-associated muscle wasting (81)

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lot 13), diluted 1:1,000 (FLAG/RPS6) or 1:10,000 (GAPDH)

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