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Haloperidol-induced PD mice models (Swiss albino mice) were utilized for the in vivo assessment through intranasal administration of the prepared nanoparticles

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Conclusion: Distinct patterns of HBV integration were observed in patients with and without HCC, providing valuable insights into the mechanisms underlying HBV integration and its association with HCC occurrence

Further research also needs to confirm the direct molecular link between CuPDTC-induced proteasome inhibition, apoptosis induction, and structural protein cleavage

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Previous research has shown that GPx1 is an effective antioxidant enzyme that cannot be replaced by any other selenoprotein to protect against generalized oxidative stress [74,75,76]