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G.BeckwithH

Since chemotherapy-induced side effects are frequently also caused by mitochondrial ROS, such as cardiotoxicity or kidney injury after doxorubicin and cisplatin treatments, respectively, these potential redox therapies should be carefully directed to the target cells

Upon exposure to n-3 PUFAs, Th17 cells generated reduced quantities of proinflammatory cytokines like IL-17, IL-22, and IL-23, whereas Treg cells demonstrated elevated production of anti-inflammatory molecules such as Foxp3 (44)