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Cisplatin treatment upregulated the expression of P66Shc in vivo and in vitro and promoted its mitochondrial translocation to exacerbate ferroptotic cell death and induce cisplatin-induced AKI [173]

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Later, intestinal IgA targeting the filaments creates negative selection pressure, making Candidalysin a key driver of intestinal symbiosis and overturning the traditional view that filaments are unfavorable for intestinal colonization (Liang et al., 2024)

PubMed Arjinpathana N, Asawanonda P

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