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and (ii) proliferative astrogliosis, which is anisomorphic with loss of domain, substantial structural reorganisation and can either be diffuse or can result in the formation of new compact limitans borders around overt fibrotic tissue lesions (Fig

This combination is commonly referenced in laboratory and preclinical research examining peptidereceptor interactions and biological signalling pathways in controlled research environments

Furthermore, clinical evidence implicates ApoE4s interaction with A is related to increased Tau deposition in AD vulnerable regions in the brain (Therriault et al., 2021), which mirrors the importance of A in the acceleration of AD Tau dynamics (Lee et al., 2022)

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