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The tissue distribution of the mRNA of ghrelin and subtypes of its receptor, GHS-R, in humans

An increasing body of evidence suggests that disturbances in the pulmonary mechanical microenvironment, a complex interplay of extracellular matrix (ECM) stiffness, alveolar surface tension, cellular forces, and cell-matrix interactions, play a crucial role in the pathogenesis and progression of these diseases ( Owing to their unique size and highly tunable physicochemical properties, nanomaterials present unparalleled opportunities to finely adjust the pathological microenvironment within the lungs ( Here, we explore how nanomaterials can address dysregulation in the lungs mechanical microenvironment by modulating ECM properties, normalizing alveolar surface tension, and influencing mechanotransduction pathways in pertinent lung cells

Byrne and Ruddle (1989) Proc

10.1186/s12931-016-0343-6 95 PengD.FuM.WangM.WeiY.WeiX