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It prevents and reverses intestinal fibrosis through suppressing myocardin-related transcription factor (MRTF) synthesis, attenuating p38 MAPK phosphorylation, and enhancing autophagic flux in fibroblasts [97]

doi: 10.3389/frfst.2024.1448148 Received 12 June 2024 Accepted 03 September 2024 Published 18 September 2024 Volume 4 - 2024 Edited by Hayriye Sebnem Harsa, Izmir Institute of Technology, Trkiye Reviewed by Oluwafemi James Caleb, Stellenbosch University, South Africa Mohini Prabha Singh, Punjab Agricultural University, India Updates Copyright 2024 Alwazeer, Hancock, Russell, Stratakos, Li, idem, Engin and LeBaron

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While fusion is generally thought to be protective in times of stress by allowing content mixing and sharing of healthy mitochondria to counteract damage to others, it has also been shown to increase the spread of detrimental mtDNA in a model of sepsis, suggesting it may exacerbate certain inflammatory conditions