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Psoriasis is inflammation of the skin

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interactions are pharmacologic co-exposure

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Detoxification of harmful lipid peroxides (Lipid-OOH) to their corresponding hydroxides (lipid-OH) by glutathione peroxidase 4 (GPX4) and glutathione-S-transferases (GST)

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Thus, the R-loop-mediated replication fork stalling observed upon HRASV12 expression may be driven by ROS-induced dissociation of the TIMELESS-TIPIN complex from the replisome, resulting in replication slowdown (Fig

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Integrative network analysis of the metabolomics and clinical data was performed using xMWAS software (v ] SerpinA3, ApoB, collagen alpha, tight junctions than HC reflecting intestinal damage

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Nutr Metab Cardiovasc Dis 24(8):808814

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