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466 CurriuMCarrilloJMassanellaMRigauJAlegreJPuigJet al

145 These findings suggest that mTORC2 plays a supportive role in glucose metabolism upon GPCR activation and that this role may be functionally distinct from classical insulin/PI3K/AKT signaling

While immortalized MSCs overcome replicative senescence, they frequently demonstrate altered paracrine secretion patterns, reduced immunomodulatory function, impaired multilineage differentiation capacity

The subsequent procedures are described in a previous study 23

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