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Several potential mechanisms have been proposed, including chelation by reductants (such as flavins or NADPH), direct electron transfer via reductases (e.g., STEAP3), and lysosomal degradation during ferritinophagy [116]

[380] [381] Fabry disease The primary treatment for ocular symptoms arising from Fabry disease, caused by alpha-galactosidase A deficiency, involves using agalsidase alpha or beta (Fabrazyme)