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Sodium channel ENaC mediates IL-17 synergized high salt induced inflammatory stress in breast cancer cells

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For instance, interventions aimed at restoring the function of antioxidant enzymes, such as superoxide dismutase 2 (SOD2), have shown promise in protecting retinal cells from the long-term damage caused by hyperglycemia-induced metabolic memory (24)

Threading through all of these complications is one quiet constant: glutathione depletion

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