Constitutive overexpression of Nrf2-dependent heme oxygenase-1 in A549 cells contributes to resistance to apoptosis induced by epigallocatechin 3-gallate
Simultaneously, glutamine metabolism supports fibrotic progression by maintaining bioenergetic and biosynthetic demands of activated myofibroblasts through ATP generation and provision of -ketoglutarate precursors [20]
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Cochrane Database Syst Rev (12)
Moreover, MDA promotes the aberrant aggregation of -syn by modifying its 15 lysine sites, thereby initiating Lewy body formation and driving the pathological process of PD (90)
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