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doi: 10.1016/j.eururo.2018.12.022 12 NohSGoAKimDBParkMJeonHWKimB

abcam), which has been known to induce AD-like astrocyte reactivity in vitro [27] upregulated the expression of candidate enzymes Putrescine Acetyltransferase ( Sat1 ), SIRT2 ( Sirt2 ) and ALDH1A1 ( Aldh1a1 ) involved in the putrescine-to-GABA degradation pathway (Fig

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In: Caballero B (ed) Encyclopedia of human nutrition

Therapeutic treatment can encourage the replacement of damaged and lost neurons from available neural stem cells, encourage the formation of new functional synaptic connections among existing neurons, and facilitate cerebral blood flow and neuroprotection

Hence, cysteine fitness constitutes a relevant mechanism of chemoresistance for cancer cells accounting for their capacity of escaping from the action of oxidative and alkylating anti-cancer drugs (93, 94, 100106)