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Mitochondrial dysfunction generates free radicals, which exacerbates oxidative stress (100)

Studies have demonstrated that alpha-lipoic acid can preserve tight junction integrity [231] and reduce neuroinflammation by inhibiting nuclear factor-kappa B (NF-B) signaling, a key pathway involved in the production of pro-inflammatory cytokines [233]

and hence, can displace them with their catechol group, which then attaches with the binuclear copper ions in the catalytic site of tyrosinase 88

These cells are involved directly through phagocytosis and the generation of oxidative radicals and enhance recruitment through the generation of inflammatory cytokines

In the absence of a liver transplant, the APAP-associated acute liver failure patients who met the KCH criteria reportedly have 8090% mortality

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