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Cystine deprivation does not promote mitochondrial oxidative stress To confirm that the persistence of respiratory function in NSCLC was not associated with mitochondrial stress as has been previously reported in other contexts 3,15,22,23 , we interrogated the mitochondrial redox state in response to cystine starvation

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The top 3 negatively enriched HALLMARK terms in high IGF-1 subgroups were epithelial mesenchymal transition, KRAS upsignaling, and allograft rejection ( Figure 8C ), and the top positively enriched HALLMARK terms were MYC targets V2, reactive oxygen species pathway, oxidative phosphorylation, and DNA repair ( Figure 8D )