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Still, these data broadly indicate a robust persistence of mitochondrial respiration under cystine deprivation that occurs independent of an expected insult to the IMM 15,22

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These microbial interactions may promote the development and progression of GC (Petra et al., 2017)

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These experimental designssuch as treating myotubes with uremic serum or specific toxins like indophenol sulfate and inducing CKD in mice or rats via 5/6 nephrectomy or adenine dietprovide causal evidence elucidating the roles of chronic inflammation, uremic toxins, and metabolic dysregulation in driving muscle wasting