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Furthermore, in addition to OXT regulation, a lipopolysaccharide (LPS)-induced depression-like behavior mouse model demonstrated that inhibiting CD38 improved neuroinflammation, microglial activation, synaptic defects, and Sirt1/STAT3 signaling, thereby improving depression-like behavior [13]

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Conclusion This case highlights the diagnostic complexity of pernicious anemia, particularly when overlapping nonspecific symptoms and comorbidities, such as fibromyalgia, hypothyroidism, and autoimmune diseases, obscure the clinical picture and delay recognition