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A comprehensive evaluation identifying contributing factors helps determine whether PTD-DBM alone is sufficient or whether additional interventions are needed

Since data on the role of astrocytic mitochondria in PD is still sparse [reviewed in (76, 95)] further experiments will be required to assess how astrocyte metabolism as well as astrocyte-neuron coupling may contribute to the development of the movement disorder

We investigated the potential role of inflammation in dACC glutamate abnormalities detected by raised peripheral cytokines and depletion of MRS glutathione, the brains principal anti-oxidant, depleted by reactive oxygen species generated by inflammation

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