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10.1016/j.immuni.2013.01.009 68

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This molecule is crucial for maintaining cellular redox homeostasis and regulating the cell cycle, including apoptosis, immunological defense, and pathological abnormalities (1,2)

The activation of NRF-2 in liver suppresses the synthesis of fatty acids [70], as observed in this study that included experimental feeding with 8-week-old NRF-2 null mice, and wild-type mice fed experimentally, observing the mRNA of sterol regulatory element-binding protein 1-c and fatty acid synthase as highly expressed in NRF-2 null [71], concluding that the protective action is afforded by the increase of AR