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The NLR family pyrin domain-containing 3 (NLRP3) inflammasome complex recognizes microbial and oxidative stress signals, such as PAMPs, ROS, and DAMPs, through its PRR

Interleukin-2 at the crossroads of effector responses, tolerance, and immunotherapy

Those mutations are found in 10 to 20 percent of all cancers, and the mutations render the gene inactive. Based on this association, the researchers hypothesized that mice lacking Arid1a would develop liver damage and, eventually, liver cancer

This patient's hyperacute 30-minute timeline to death and the lack of neutrophilic infiltrates on autopsy make bilateral bronchopneumonia unlikely

Key mechanisms further elaborate this connection