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Secondly, although we controlled for current antipsychotic medication intake and metabolite concentrations, possible confounding effects of long-term medication use should be considered

Hutchinson-Gilford Progeria Syndrome: An Overview of the Molecular Mechanism, Pathophysiology and Therapeutic Approach

The duration and route of treatment should be determined by the patient presentation

The relationship between sensitivity and 1specificity for SOD and GPx were assessed to find the ROC pattern of each enzyme (Figure 1)

TPP thiamine pyrophosphate Boosting TPP in cells stressed by high glucose concentrations results in the opening of a safety valve in the normal metabolism of blood sugar through an enzyme known as transketolase

These unstable molecules can oxidise other cellular biomolecules, resulting in oxidative stress